Anandamide

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Anandamide also is known as N-arachidonoylethanolamine or AEA, is an endogenous cannabinoid neurotransmitter. The name is taken from the Sanskrit word (and Hinduistic religious term) ananda, which means "joy, bliss, delight", and amide. It is synthesized from N-arachidonoyl phosphatidylethanolamine by multiple pathways. It is degraded primarily by the fatty acid amide hydrolase (FAAH) enzyme, which converts anandamide into ethanolamine and arachidonic acid. As such, inhibitors of FAAH lead to elevated anandamide levels and are being pursued by therapeutic use.

Anandamide's effects can occur in either the central or peripheral nervous system. These distinct effects are mediated primarily by CB1 cannabinoid receptors in the central nervous system, and CB2 cannabinoid receptors in the periphery. The latter is mainly involved in the functions of the immune system. Cannabinoid receptors were originally discovered as being sensitive to Δ9-tetrahydrocannabinol (Δ9-THC, commonly called THC), which is the primary psychoactive cannabinoid found in cannabis.

The toxicity and long-term health effects of recreational Anandamide (AEA) use do not seem to have been studied in any scientific context and the exact toxic dosage is unknown.

Formula: C22H37NO2

CAS Number: 94421-68-8

Molecular Weight: 347.53

Purity: 99.8% 

Appearance: Powder

Research Chemicals are available in Crystalline solid, crystal, powder, and liquid form

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